Answering a Question from a FB Reader
Intended or unintended consequences? The second is unprecedented misfeasance. The first is genocide.
If it was a one time thing (a transient transfection), junk proteins, whether self or not-self, get cleaned up quickly, especially not-self proteins.
However, if it’s a stable transfection, possible if cellular reverse transcriptase makes DNA copies of the viral mRNA and inserts the DNA copies randomly into the nuclear DNA of that particular cell, then the daughter cells produced from the mitosis of that cell will carry that DNA-encoded viral mRNA throughout succeeding generations of that particular cell line in whichever organ it exists.
At certain points in time, some biological trigger, who knows what and who knows when, will cause the transcription machinery in the nucleus to make mRNA copies of that DNA-encoded viral gene. Those mRNA molecules will be transported from the nucleus into the cytosol where ribosomes will automatically translate them into the viral spike protein.
The amount of that spike protein will depend on how many copies of the viral mRNA have been produced and how many times they’ll be run through the ribosomes until they’re degraded by housekeeping proteins inside the cell.
This is what happens with Herpes simplex and Herpes zoster. In their case, it was the entire viral genome that was reverse transcribed into a DNA-encoded form. The lesions produced (from shingles or a fever blister on the lips or elsewhere) will shed actual viruses that are as infectious as the originals.
In the case of spike protein, those cells producing it from newly transcribed viral mRNA will detect both viral mRNA and the viral protein and alert the innate immune system to launch innate immune inflammatory attacks in an attempt to kill those cells. A lot of innocent bystander cells get whacked along with them.
And this is the principal danger of the viral mRNA products each time they are used. They are indiscriminate and will infect any cell of any type in any organ they bump into with viral mRNA, inevitably provoking the innate immune inflammatory attacks.
The degree of those attacks depends on how many cells are responding and how many different organ systems are signaling.
If someone survives that, they may have enough inflammatory damage to suffer dysfunction in those organs. Subsequent exposure to more viral mRNA products can exacerbate that damage. Organs suffering sufficient damage may no longer be able to keep you alive.
But if you survive and you never get another viral mRNA shot, then you still have the possibility of having had a variety of cells in multiple organs via cellular reverse transcriptase insert a DNA-encoded version of that viral gene into the cell’s nucleus.
The longer you live without the transcription of that embedded gene being triggered to produce the viral mRNA, the more daughter cells there will be in that organ, meaning that when the triggering event happens, it will trigger the production of new viral mRNA in the descendants of the original cell. And since it could have happened in cells of multiple organs, the triggering is not likely to be the same, but something specific to that cell type, meaning the possibility of different organs at different time triggering innate immune inflammatory attacks.
The way it will appear to a clinician will be the appearance of an autoimmune condition typical for that organ.
Here’s what you could expect to see in the wake of the use of these viral mRNA products:
1. An increase in what will be supposed to be autoimmune conditions,
2. An increase in multiple autoimmune conditions in single individuals.
3. The appearance of this at rates and to degrees significantly higher that what was seen prior to 2021.
4. The appearance of this in recipients of the viral mRNA products but not in the product-negative population of the world, meaning that once the general population is not used to calculate the incidence, the rate will be seen to become much higher relative to pre-2021 and the product-free population.
5. The appearance of this around the globe in a manner consistent with the dates of introduction of the viral mRNA products in their countries.
6. The appearance of this within any particular country in a manner consistent with the dates of various groups given priority targeting to receive the viral mRNA products.
7. An increase in affected populations that’s dose dependent with the number of viral mRNA treatments.
8. An increase of all incidents of this with the passage of time consistent with the rate of increase of daughter cell population.
9. An increase of all incidents of this in 8 above but differentially with the rate of increase in daughter cell population of different cells types with different rates of proliferation: meaning that you could expect to see it the most in epithelial cells, cells with a very high rate of turnover such as those arising from stem cell populations in bone marrow and elsewhere supplying the various types of blood cells and immune system cells, certain organs with high metabolic activity, in the rapid proliferation of all cell types seen during development from conception to adulthood, especially in puberty, and especially during pregnancy.
Many of these have already been noted over the past three and a half years. It will continue until the populations of affected daughter cells in one or more organ systems will become large enough to provoke an innate immune response too great to survive.