One of two responses to yet another of a certain person’s lazy, incoherent, knee-jerk rants on the origin, nature, and means of action of the viral mRNA products.
________
So far now, even in recent papers about excess mortality, they ALL miss the most fundamental facts about those products, things that are among the first to occur and that literally drive all the rest of the effects that are the basis of all adverse events and excess death:
1. They all rely on absolutely trivial means to introduce viral genes into healthy cells to drive, from inside the cells, the production of viral proteins that
A. have no business being there,
B. have no means of entering there on their own, and
C. have a history of being produced there solely as the result of viral infection; have a history of being detected there solely as the result of viral infection; have a predictable effect both on the cell and the innate and adaptive immune system in the context of viral infection.
Those products, therefore, are the functional equivalent of viruses with the predictable outcomes of viral infections with some important variations.
2. The reason that people are talking about the constituents of the products’ delivery vehicles as being the cause of adverse events and sudden and/or excess deaths is only because they have completely overlooked the actual cause (see 1) and are basically scratching their heads, saying, “Well, it’s got to be those constituents because what else is there?”
Well, here’s what: there’s a crapload of cells that are being infected with viral genes in a way that (with the exception of the AstraZeneca product) has no parallel in nature and to a degree that also has no parallel in nature except at the end of a runaway viral infection that the combined efforts of the innate and adaptive immune systems have been unable to put down.
Their approach to the “problem” is pretty much the equivalent of someone attributing death due to multiple stab wounds to toxic effects of the metal from which the knife was fashioned or to residues left on the knife from the manufacturing process. No, it’s the puncturing of organs and the dissection of major blood vessels caused by action of the knife itself, not the components of the knife or contaminants on it.
All cause mortality are all the deaths coming as the result of every possible thing that can go wrong badly enough in every major organ system. There may be multiple things going on that can cause those deaths and many of them may be specific to various organ systems, the damage and death being attributed to “cardiovascular disease” or to “pulmonary disease” or to “kidney disease,” the causes of which may be genetic or viral or bacterial or toxins or neoplasms or “autoimmune” attacks.
And because of that variety of specific causes in different organ systems, the manufacturers of the viral mRNA products attempt to dodge responsibility by dishonestly claiming, “But vaccines don’t have such widespread effects in multiple organ systems and our products are only vaccines.”
Dishonest because A. their products are not and have never been vaccines, and B. they know it.
Others may claim that, since the many and varied conditions responsible for all cause mortality have a wide variety of specific causes, there can’t be a single cause to account for an increase in all-cause mortality.
And the reason they say that is because they’re limiting themselves to all the typical causes of those deaths and are overlooking the most important, common factor of all those deaths that have occurred over how many million years people have been around.
That common factor is this: something occurs that messes up organ function to the point that life can no longer be sustained.
Basically, it doesn’t matter WHAT that is only THAT it messes up function enough to cause organ failure (or enough organ impairment) which then leads to death. Sure, some of those causes are different based on the organ involved and what sorts of things can specifically mess it up.
But think of it like this: you can kill someone by sticking a pencil into his brain or into his heart or by puncturing his lungs or perforating his intestine; but NO one would describe those deaths as central nervous system disease or cardiovascular disease or pulmonary disease or GI disease.
So what’s one cause of disease and dysfunction in multiple organ systems?
Inflammatory damage.
And what initiates the inflammation?
It’s not overuse or physical trauma. It’s not viral or bacterial infection. It’s the innate immune response TO those insults, the purpose of which is either to attack pathogens or to initiate wound repair.
Can that innate immune response get out of control and cause additional damage, dysfunction, even death?
Yes. That is well-known.
So, if you get a respiratory infection, you generally suffer some specific types of inflammation. You get a urinary tract infection and have inflammatory responses in that region. You may go to a dentist and, as the result of the typical trauma, get certain oral bacteria introduced into the blood stream that can cause infection of cardiac tissues, recruitment of the innate immune system, inflammation, and possible long term damage of the heart or even death.
And, basically, because there is NO living part of your body that is not supplied by the circulatory system or that does not share the extracellular fluid compartment or that is not connected to the lymphatic system, then this means that everything is open to both the innate and adaptive immune systems.
And since the innate immune system, given sufficient provocation, can initiate innate immune inflammatory attacks, then there is literally almost nowhere in your body that cannot be subjected to innate immune-driven inflammation and, given sufficient provocation, innate immune inflammatory attack. But if you get the inflammation of tennis elbow or the inflammation of a sore throat or the inflammation around a cut on your big toe, you don’t suffer inflammation everywhere.
To suffer widespread inflammation or widespread inflammatory attacks you need a widespread cause. And that is usually widespread dissemination of viral or bacterial pathogens. Or someone could shoot you up intravenously with a big load of Freund’s adjuvant. Given sufficient provocation, your innate immune system can become your own worst enemy. In addition, given deliberate provocation, your innate immune system can be turned into the most intimate and ubiquitous weapon and used against you.
So now let’s think of what would have to occur for this to happen.
Though some diseases like Spanish flu or smallpox can trigger massive, deadly innate immune inflammatory attacks, most infections are local and can be dealt with locally by the innate immune system to prevent a more global escalation into a deadly systems-wide innate immune inflammatory response.
And one reason that most viral attacks are limited is because they rely on being able to manipulate specific cell surface receptor to gain entry to start replication. If you don’t have that cell surface receptor, then you will NEVER get infected by that particular virus.
How do we know this?
In at least three ways:
A. There is a specific cell surface receptor that Yersinia pestis (NOT a virus) uses to gain entry to cells expressing it to reproduce, the clinical presentation of which is the Black Death. People who had a mutation in that cell surface receptor were immune to the Black Death and their descendants expressing that mutated cell surface receptor are immune to HIV (a virus) that relies on that receptor to gain entrance to replicate inside the cell.
B. The occasional appearance of respiratory viruses that made the leap from animal to human host by the chance mutation of viral coat proteins into a form that can manipulate a specific cell surface receptor to gain entry to the specific subset of cells expressing that receptor to start replication.
C. The point of experiments in gain-of-function research. This is the direct approach to B. The object is to alter certain viral coat proteins amino acid by amino acid until the protein sequence is able to activate a specific cell surface receptor to gain entry for the virus into the cell to use its cellular machinery to replicate.
It’s not difficult to imagine what would happen if a virus were able to gain access to the largest population of cells expressing a specific cell surface receptor. The result would be widespread infection and a greatly heightened innate immune response.
Or this. What if there were specific populations of cells in specific organs with a specific cell surface receptor. A virus with the viral coat proteins able to manipulate just those receptors on just those cells would show up as an infection of that organ and provoke an innate immune inflammatory attack on that organ.
So what if some virus were to come on the scene that could manipulate the specific cell surface receptors of ANY cell to gain entry to replicate, like it was the Swiss Army knife of viral infection. And when those cells start to detect the presence of viral mRNA and viral protein inside themselves and start signaling the innate immune system, “We’ve been compromised by viruses now in replication mode. Come kill us,” will the result be negligible or catastrophic?
Answer: pretty frigging catastrophic.
But, hey, what a relief, right, that there is no virus with universal access to all cell types?
Talk about dodging a potentially world-ending bullet!
But what if the virus didn’t even need a specific cell surface receptor to get into any cell it bumped into? And what if it couldn’t be detected by the innate immune system on its way to its next cell for more Joy of Replication?
What would you see? You’d see large levels of innate immune inflammatory attacks on the tissues of multiple organ systems.
You’d see tissue and organ dysfunction.
You’d also see an increase in all cause mortality.
Why?
Because the virus is no longer limited in its choice of targets.
So, again, thank God there are no such indiscriminate, undetectable, universal viruses!
But consider this: the viral mRNA products in use can, undetected, get into any cell they bump into in any organ.
Once in, their cargo of copies of a single viral gene is used to make many copies of viral proteins.
Those product-compromised cells detect the presence of viral mRNA and viral protein and give the alert to the innate immune system to launch inflammatory attacks on them wherever they are found in a wide variety of organ systems.
Given this, here what you could expect to see:
1. Increases in deaths due to cardiovascular and pulmonary compromise by inflammatory attack, some of them sudden.
2. Increases in all cause mortality because the innate immune inflammatory attacks are against all organs with compromised cells. If it goes on long enough and is repeated enough times, those organs will fail also.
3. You would see the increase in all cause mortality in product-compromised versus product-free individuals.
4. You would see it across all age groups but especially in those who are already standing on a banana peel on the edge of a cliff of existing comorbid conditions.
5. You would see the increase in all cause mortality around the world coincident with the products’ date of introduction and also, group by group, among those who were first subjected to it.
6. Given the sensitivity to disruption of developmental events, you would see an increase in miscarriages among the product-plus population and in children.
7. You would see the companies scrambling to say, “It can’t be us because our products are vaccines, and vaccines don’t do that.”
So, have we seen 1-7 over the past 3.5 or so years?
Yes.
And now you know who to blame.