The Skank or the Tiger?

The Skank or the Tiger?

Door Number 1 or Door Number 2?

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No, Manny, you’re still missing the point.

Here’s the first effect seen from healthy cells in multiple organ systems being infected by ONE COVID-19 gene is the same effect as healthy cells being infected by the COVID-19 virus:

An innate immune inflammatory attack that is initiated by the infected cells as soon as they detect within themselves the presence of COVID-19 viral mRNA and COVID-19 viral protein.

As far as initiating the innate immune response is concerned, it makes NO difference whether it’s ALL the viral mRNA molecules of an entire COVID-19 viral genome and ALL the resulting COVID-19 viral proteins (as happens during a normal infection in certain cells in the respiratory tract) or the mRNA for single COVID-19 gene and the resulting viral protein for that ONE gene (as happens during a deliberate infection of cells using a viral mRNA product).

Why?

Because the minimum necessary condition required for that innate immune response is the presence of viral mRNA and viral protein detected INSIDE healthy cells by those same cells.

So what makes it a big deal when they do it via a viral mRNA product containing a single, chimeric viral gene?

Because it’s taking place, NOT just in a few ACE2 receptor-expressing cells in the respiratory system, but in any cells those particles of product bump into in any organ of the body all at once.

To the innate immune system, that looks like a sudden, massive viral infection compromising many organs simultaneously.

The result is not going to be a sniffle and some nasal congestion as in the early stages of a common cold.

It’s going to be widespread and damaging innate immune inflammatory attacks.

It is THOSE attacks that do the damage, not the virus (or single viral gene) initiating them.

But worse.

Why?

Because the folks who engineered that included the sequence for a viral epitope known to cause the innate immune system to crank up a much more aggressive response against the viral protein.

That response comes later, after the one described above.

Why later?

Because as long as the viral protein is still inside the cells compromised by the viral mRNA product, the innate immune system cannot directly detect it, only via the sensors inside the compromised cells that detect the presence of viral protein.

After those cells are hit by innate immune inflammatory attacks called in to kill them, the viral protein inside is released along with the cellular debris. NOW the direct contact of the innate immune system components with the viral proteins initiates a second wave of innate immune inflammatory response.

But because that viral protein now contains an extra viral protein epitope from another viral pathogen, an epitope that greatly increases the level of innate immune response beyond what it would have been for the basic viral spike protein, there is another great escalation of innate immune response on top of the first.

So, here are your two choices.

Door Number 1: You could maybe get infected by the COVID-19 virus in a relatively small number of just one particular type of cell in just one organ system.

Or

Door Number 2: You can get shot up with the viral mRNA product with the certainty that many cells in many different organs of your body will get infected with one COVID-19 gene and all start signaling about the same time, “I’m infected by a virus. Come kill me.”

I’d rather risk the possibility of getting infected by a relatively innocuous common cold virus that over half the population has already been resistant to for many decades than to get shot up with something that was designed to infect any cell of any organ in a way my immune system can do nothing to prevent—EVER.

But that’s not the worst part.

After Number 2 happens to you, the extra surprise grafted onto those proteins is detected. And the initial much too large innate immune response is followed by even greater innate immune response throughout multiple systems.

And there is ZERO reason why those designing this would not have known that would be the inevitable outcome.

Why?

Because it’s just the intersection of basic immunology with the completely bizarre off-label use of cell transfection reagents to infect with viral genes any cell they come into contact with.

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Gee, that doesn’t sound like a vaccine that prevents infection. It sounds more like something designed to infect you.

And, Manny, after this occurs, if you’re given an antibody test, you’ll test positive. That doesn’t mean the viral mRNA products “gave you COVID-19.” No, you just developed a predictable adaptive immune response against just ONE COVID-19 viral protein.

What they gave you is much, much worse:

The viral mRNA products constitute an artificial virus capable of infecting any cell at all in any organ with all the completely predictable results but to a much greater degree, throughout a much greater volume of your body, and then, after they do that to you, they point to the effects and say that means you have to keep coming in for “boosters” that will do the same and more.

Why more?

For at least two reasons.

1. Everything you suffered the first time, you’ll suffer again.

2. Organs damaged the first time will suffer cumulative damage with each additional shot. That is, you will be suffering multiple and increased levels of comorbidity.

Guess what the single largest predictors of death by COVID-19 is.

It’s your number and severity of comorbid conditions.

Guess what the single largest predictor of death following getting shot up with viral mRNA products is.

It’s your number and severity of comorbid conditions.

3. The reason they tell you that you need to get a “booster” is because the spike proteins mutate, meaning that antibodies and T cells against one won’t work against “new” ones.

Yeah, but one viral antigen doesn’t have a single epitope. If that one protein were so heavily mutated that all the epitopes were changed, then that spike protein would no longer be able to activate the ACE2 receptor to bring in the virus for replication.

Fortunately, for the viral mRNA companies, they don’t have to worry about that because their products don’t have to make it past a cell surface receptor gatekeeper. Unlike a virus, they just go right through the membrane and start making spike protein.

But even though the companies give you the mRNA for a slightly different spike protein, one thing remains the same: that additional bit from another virus that potentiates the innate immune response.

The reason something is called a booster is because it boosts your adaptive immune response to a particular antigen. This is why the number and severity of colds you get drops off as you get out of childhood into your teens. Each cold helps to build your immune system response. After a while it is such that the pathogens are so quickly taken out at such an early stage that you don’t even know you’ve been reinfected.

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But what happens with the viral mRNA products?

They will consistently produce large quantities of viral antigen.

If you get repeated exposure to a variety of so-called “boosters” but they all have that same extra bit to potentiate innate immune response, you are boosting your adaptive response to that bit.

So what’s the big deal?

In a normal viral infection by the same virus, as your antibodies and T cells increase, the more greatly reduced your innate immune response.

Why?

Because at earlier and earlier timepoints with each subsequent infection those pathogens are being prevented from infecting more cells. This reduces the damaging innate immune inflammatory attacks. Those attacks depend on the number of viral proteins encountered by the members of the innate immune system.

But that’s not the case with viral mRNA products. They are still going to infect just as many cells as they always do. They are going to produce just as many spike protein molecules as they always do. As a result, not only will each dose of viral mRNA product result in large innate immune inflammatory attacks, it will increase the number of antibodies and T cells against that extra epitope tag.

So you’d think, well, no big deal, because they will just more quickly bind to those spike proteins.

But those spike proteins have an additional property. They can embed themselves in the cellular membrane.

So?

Well, guess what an otherwise healthy cell with a viral protein embedded in its membrane looks like?

It looks like a cell in which viral replication is taking place.

And when an antibody binds to it, it is marked for destruction either by a killer T cell or by innate immune inflammatory attacks.

So guess what’s going to be happening when you’ve built a big adaptive immune response and your body keeps getting flooded repeatedly with large numbers of viral proteins that can embed themselves in cells of organs throughout your body?

You are going to be suffering repeated and compounding innate immune inflammatory damage.

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Now, in the case of a virus, viral proliferation is halted at increasingly earlier time points following infection, meaning decreasing levels of innate immune inflammatory attacks. In this situation, there is a limit to infection that is reached more and more quickly. It may look like you’re not getting reinfected, but that’s only because the threat is taken out before you can feel the effects of the innate immune system fighting it.

But in the case of the viral mRNA products, that never happens because, unlike viruses, they can’t be spotted and taken out on their way to the cells they’re going to infect. They’re going to make it to the worst possible place to do the worst possible thing as though they were invisible viruses. In this situation, there is no limit to infection because they can’t be detected and there’s no limit to reinfection as long as you keep going back for more “boosters.”

In the end, the only limit to reinfection is like what happens with some of the really bad viruses: that’s when you’ve accumulated so much innate immune inflammatory damage that you die.

Final thought:

When you die from a respiratory virus, where will people see the inflammatory damage that led to your death? Mostly in the lungs.

When you die from a virus that affects mostly your liver, like hepatitis, where will people see the inflammatory damage that led your death? Mostly in your liver.

When you die from a virus that can infect your heart, where will people see the inflammatory damage that led to your death? Mostly in your heart.

So if you’re getting shot up with something that will provoke innate immune inflammatory attacks in any or in multiple organ systems, where will people see the inflammatory damage that led to your death? In all sorts of places.

And if you’re already suffering from multiple comorbid conditions and you get shot up with something that will provoke innate immune inflammatory attacks in any or in multiple organ systems, where will people see the inflammatory damage that led to your death? In those different places affected by your comorbidities.

Now, guess what distinguishes these two populations since the beginning of 2021:

1. The one third of the human race who have never received a viral mRNA product.

2. The two thirds of the human race who have received between one and six doses.

All cause mortality for those in 1 has remained about the same as it was in 2020 (which had a lower mortality rate than 55 of the last 60 years).

All cause mortality for those in 2 has significantly increased since the beginning of 2021, and it has increased in a dose-dependent manner with the number of viral mRNA injections.

So, Manny, you’re worried about entirely the wrong thing.

What you should be worried about is that this product was created and that its creators say, “All that stuff can’t be our fault because our vaccine only gives your body the information it needs to build immunity; besides, vaccines don’t act like that.”

Yeah, but you know what does act like that?

A product that they call a vaccine but that behaves like an indiscriminate, multisystemic, undetectable, one-gene virus against which no one will EVER develop an adaptive immune response.